A common tactic of creationists, writing about ERVs, (see this page) is to suggest that ERVs were designed into the genomes of different "kinds" of organisms to provide a means of distributing useful genes by exogenization and re-endogenization. So which were the "original, designed-in" ERVs, and which were the result of exogenization and re-endogenization? How does "design" explain that the latter types are still in orthologous locations in different species? As we know, integrase is incapable of targetting specific DNA loci.
If anyone thinks that ERVs are designed of a purpose or for several purposes, they must have answers to the following.
a) What is reverse transcriptase designed to do?
b) What is integrase designed to do?
c) Why were ERVs designed with a viral codon bias?
d) What is the design purpose of re-transcribable promoters?
e) What were the HERVs that generated the consensus sequence that generated Phoenix designed for?
f) What is the design purpose of both exogenous and endogenous KoRV?
g) If chimps and humans have commonly located ERVs, what is the design purpose of giving these common ERVs common disabling mutations?
h) What is the design purpose of giving some people HERVs and not others?
i) What is the design purpose of creating different syncytins in different placental lineages?
Someone attempted to answer these questions. I reply here.
You see, it's no use just waving your arms in the air and saying, "Design". Endogenization explains all these features. How does "design" explain them?